<?xml version="1.0" encoding="UTF-8"?>
<!DOCTYPE article PUBLIC "-//NLM//DTD JATS (Z39.96) Journal Publishing DTD v1.3 20210610//EN" "JATS-journalpublishing1-3.dtd">
<article article-type="research-article" dtd-version="1.3" xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance" xml:lang="ru"><front><journal-meta><journal-id journal-id-type="publisher-id">porozendo</journal-id><journal-title-group><journal-title xml:lang="ru">Остеопороз и остеопатии</journal-title><trans-title-group xml:lang="en"><trans-title>Osteoporosis and Bone Diseases</trans-title></trans-title-group></journal-title-group><issn pub-type="ppub">2072-2680</issn><issn pub-type="epub">2311-0716</issn><publisher><publisher-name>Endocrinology Research Centre</publisher-name></publisher></journal-meta><article-meta><article-id pub-id-type="doi">10.14341/osteo2016113-14</article-id><article-id custom-type="elpub" pub-id-type="custom">porozendo-8931</article-id><article-categories><subj-group subj-group-type="heading"><subject>Research Article</subject></subj-group><subj-group subj-group-type="section-heading" xml:lang="ru"><subject>Статьи</subject></subj-group><subj-group subj-group-type="section-heading" xml:lang="en"><subject>Articles</subject></subj-group></article-categories><title-group><article-title>WNT СИГНАЛЬНЫЙ ПУТЬ В ИССЛЕДОВАНИЯХ КОСТНОЙ ТКАНИ</article-title><trans-title-group xml:lang="en"><trans-title>WNT-SIGNALING PATHWAY IN THE BONE TISSUE STUDIES</trans-title></trans-title-group></title-group><contrib-group><contrib contrib-type="author" corresp="yes"><name-alternatives><name name-style="eastern" xml:lang="ru"><surname>Белая</surname><given-names>Ж Е</given-names></name><name name-style="western" xml:lang="en"><surname>Belaya</surname><given-names>Zh E</given-names></name></name-alternatives><bio xml:lang="ru"><p>зав. отделением нейроэндокринологии и остеопатий, д.м.н</p></bio><bio xml:lang="en"/><email xlink:type="simple">janna.belaya@gmail.com</email><xref ref-type="aff" rid="aff-1"/></contrib></contrib-group><aff-alternatives id="aff-1"><aff xml:lang="ru">ФГБУ «Эндокринологический научный центр» Минздрава России</aff><aff xml:lang="en"></aff></aff-alternatives><pub-date pub-type="collection"><year>2016</year></pub-date><pub-date pub-type="epub"><day>15</day><month>12</month><year>2016</year></pub-date><volume>19</volume><issue>1</issue><issue-title>№1 (2016)</issue-title><fpage>13</fpage><lpage>14</lpage><permissions><copyright-statement>Copyright &amp;#x00A9; Белая Ж.Е., 2016</copyright-statement><copyright-year>2016</copyright-year><copyright-holder xml:lang="ru">Белая Ж.Е.</copyright-holder><copyright-holder xml:lang="en">Belaya Z.E.</copyright-holder><license license-type="creative-commons-attribution" xlink:href="https://creativecommons.org/licenses/by/4.0/" xlink:type="simple"><license-p>This work is licensed under a Creative Commons Attribution 4.0 License.</license-p></license></permissions><self-uri xlink:href="https://www.osteo-endojournals.ru/jour/article/view/8931">https://www.osteo-endojournals.ru/jour/article/view/8931</self-uri><abstract/><trans-abstract xml:lang="en"/></article-meta></front><back><ref-list><title>References</title><ref id="cit1"><label>1</label><citation-alternatives><mixed-citation xml:lang="ru">Nusse R, Varmus H. Many tumors induced by the mouse mammary tumor virus contain a provirus integrated in the same region of the host genome. Cell, 1982, 31, 99-109</mixed-citation><mixed-citation xml:lang="en">Nusse R, Varmus H. Many tumors induced by the mouse mammary tumor virus contain a provirus integrated in the same region of the host genome. Cell, 1982, 31, 99-109</mixed-citation></citation-alternatives></ref><ref id="cit2"><label>2</label><citation-alternatives><mixed-citation xml:lang="ru">Nüsslein-Volhard C, Wieschaus E (October 1980). «Mutations affecting segment number and polarity in Drosophila». Nature 287 (5785): 795-801. DOI:10.1038/287795a0. PMID 6776413.</mixed-citation><mixed-citation xml:lang="en">Nüsslein-Volhard C, Wieschaus E (October 1980). «Mutations affecting segment number and polarity in Drosophila». Nature 287 (5785): 795-801. DOI:10.1038/287795a0. PMID 6776413.</mixed-citation></citation-alternatives></ref><ref id="cit3"><label>3</label><citation-alternatives><mixed-citation xml:lang="ru">Holstein TW. The evolution of the Wnt pathway. Cold Spring Harb Perspect Biol 2012, 4: a007922</mixed-citation><mixed-citation xml:lang="en">Holstein TW. The evolution of the Wnt pathway. Cold Spring Harb Perspect Biol 2012, 4: a007922</mixed-citation></citation-alternatives></ref><ref id="cit4"><label>4</label><citation-alternatives><mixed-citation xml:lang="ru">Clevers H, Nusse R.: Wnt/β-Catenin signaling and disease. Cell, 2012, 149, 1192-1205</mixed-citation><mixed-citation xml:lang="en">Clevers H, Nusse R.: Wnt/β-Catenin signaling and disease. Cell, 2012, 149, 1192-1205</mixed-citation></citation-alternatives></ref><ref id="cit5"><label>5</label><citation-alternatives><mixed-citation xml:lang="ru">Willert K, Nusse R. Wnt proteins. Cold Spring Harb Perspect Biol, 2012, 4:a007864</mixed-citation><mixed-citation xml:lang="en">Willert K, Nusse R. Wnt proteins. Cold Spring Harb Perspect Biol, 2012, 4:a007864</mixed-citation></citation-alternatives></ref><ref id="cit6"><label>6</label><citation-alternatives><mixed-citation xml:lang="ru">Wang Y, Li Y, Paulson C, Shao J-Z, Zhang X, Wu M, Chen.: Wnt and the Wnt signaling pathway in bone development and disease. Front Biosci, 2014, 19, 379-407</mixed-citation><mixed-citation xml:lang="en">Wang Y, Li Y, Paulson C, Shao J-Z, Zhang X, Wu M, Chen.: Wnt and the Wnt signaling pathway in bone development and disease. Front Biosci, 2014, 19, 379-407</mixed-citation></citation-alternatives></ref><ref id="cit7"><label>7</label><citation-alternatives><mixed-citation xml:lang="ru">Гребенникова ТА, Белая ЖЕ, Рожинская ЛЯ, Мельниченко ГА, Дедов ИИ: Эпигенетические аспекты остеопороза. Вестник Российской Академии Медицинских Наук, 2015, Том 70, №5, стр. 541-548</mixed-citation><mixed-citation xml:lang="en">Гребенникова ТА, Белая ЖЕ, Рожинская ЛЯ, Мельниченко ГА, Дедов ИИ: Эпигенетические аспекты остеопороза. Вестник Российской Академии Медицинских Наук, 2015, Том 70, №5, стр. 541-548</mixed-citation></citation-alternatives></ref><ref id="cit8"><label>8</label><citation-alternatives><mixed-citation xml:lang="ru">Yavropoulou MP, Xygonakis C, Lolou M, Karadimou F, Yovos JG.: The sclerostin story: from human genetics to the development of novel anabolic treatment for osteoporosis. Hormones, 2014, 13, 476-487</mixed-citation><mixed-citation xml:lang="en">Yavropoulou MP, Xygonakis C, Lolou M, Karadimou F, Yovos JG.: The sclerostin story: from human genetics to the development of novel anabolic treatment for osteoporosis. Hormones, 2014, 13, 476-487</mixed-citation></citation-alternatives></ref><ref id="cit9"><label>9</label><citation-alternatives><mixed-citation xml:lang="ru">Belaya ZE, Rozhinskaya LY, Melnichenko GA, Solodovnikov AG, Dragunova NV, Iljin AV, Dzeranova LK, Dedov II.: Serum extracellular secreted antagonists of the canonical Wnt/β-catenin signaling pathway in patients with Cushing’s syndrome. J. Osteoporosis International, 2013, Vol. 24, pp. 2191-2199 (DOI: 10.1007/s00198-013-2268-y)</mixed-citation><mixed-citation xml:lang="en">Belaya ZE, Rozhinskaya LY, Melnichenko GA, Solodovnikov AG, Dragunova NV, Iljin AV, Dzeranova LK, Dedov II.: Serum extracellular secreted antagonists of the canonical Wnt/β-catenin signaling pathway in patients with Cushing’s syndrome. J. Osteoporosis International, 2013, Vol. 24, pp. 2191-2199 (DOI: 10.1007/s00198-013-2268-y)</mixed-citation></citation-alternatives></ref><ref id="cit10"><label>10</label><citation-alternatives><mixed-citation xml:lang="ru">Yao W, Cheng Z, Busse C, Pham A, Nakamura MC, Lane NE (2008) Glucocorticoid excess in mice results in early activation of osteoclastogenesis and adipogenesis and prolonged suppression of osteogenesis. Arthritis Rheum 58: 1674-1686</mixed-citation><mixed-citation xml:lang="en">Yao W, Cheng Z, Busse C, Pham A, Nakamura MC, Lane NE (2008) Glucocorticoid excess in mice results in early activation of osteoclastogenesis and adipogenesis and prolonged suppression of osteogenesis. Arthritis Rheum 58: 1674-1686</mixed-citation></citation-alternatives></ref><ref id="cit11"><label>11</label><citation-alternatives><mixed-citation xml:lang="ru">Larsson S. Anti-sclerostin - is there an indication? Injury. 2016;47(1):31-35. doi: 10.1016/S0020-1383(16)30008-0</mixed-citation><mixed-citation xml:lang="en">Larsson S. Anti-sclerostin - is there an indication? Injury. 2016;47(1):31-35. doi: 10.1016/S0020-1383(16)30008-0</mixed-citation></citation-alternatives></ref><ref id="cit12"><label>12</label><citation-alternatives><mixed-citation xml:lang="ru">McClung MR, Grauer A, Boonen S, Bolognese MA, Brown JP, Diez-Perez A, Langdahl BL, Reginster JY, Zanchetta JR, Wasserman SM, Katz L, Maddox J, Yang YC, Libanati C, Bone HG. Romosozumab in postmenopausal women with low bone mineral density. N Engl J Med. 2014 Jan 30;370(5):412-20. doi: 10.1056/NEJMoa1305224. Epub 2014 Jan 1.</mixed-citation><mixed-citation xml:lang="en">McClung MR, Grauer A, Boonen S, Bolognese MA, Brown JP, Diez-Perez A, Langdahl BL, Reginster JY, Zanchetta JR, Wasserman SM, Katz L, Maddox J, Yang YC, Libanati C, Bone HG. Romosozumab in postmenopausal women with low bone mineral density. N Engl J Med. 2014 Jan 30;370(5):412-20. doi: 10.1056/NEJMoa1305224. Epub 2014 Jan 1.</mixed-citation></citation-alternatives></ref></ref-list><fn-group><fn fn-type="conflict"><p>The authors declare that there are no conflicts of interest present.</p></fn></fn-group></back></article>
